ID   CX8_CONCB               Reviewed;         107 AA.
AC   B0L0Y6;
DT   31-OCT-2012, integrated into UniProtKB/Swiss-Prot.
DT   18-MAR-2008, sequence version 1.
DT   25-MAY-2022, entry version 16.
DE   RecName: Full=Putative conotoxin;
DE   Flags: Precursor;
OS   Conus caracteristicus (Characteristic cone).
OC   Eukaryota; Metazoa; Spiralia; Lophotrochozoa; Mollusca; Gastropoda;
OC   Caenogastropoda; Neogastropoda; Conoidea; Conidae; Conus.
OX   NCBI_TaxID=89440;
RN   [1]
RP   NUCLEOTIDE SEQUENCE [MRNA].
RA   Liu L., Chi C.W., Wang C.G.;
RT   "cDNA of a new protein precursor from Conus caracteristicus.";
RL   Submitted (JAN-2007) to the EMBL/GenBank/DDBJ databases.
CC   -!- FUNCTION: Acts as a neurotoxin. {ECO:0000250}.
CC   -!- SUBCELLULAR LOCATION: Secreted {ECO:0000250}.
CC   -!- TISSUE SPECIFICITY: Expressed by the venom duct.
CC   -!- DOMAIN: The cysteine framework is VIII (C-C-C-C-C-C-C-C-C-C).
CC   -!- PTM: Contains 5 disulfide bonds. {ECO:0000250}.
CC   ---------------------------------------------------------------------------
CC   Copyrighted by the UniProt Consortium, see https://www.uniprot.org/terms
CC   Distributed under the Creative Commons Attribution (CC BY 4.0) License
CC   ---------------------------------------------------------------------------
DR   EMBL; EF208033; ABQ00573.1; -; mRNA.
DR   AlphaFoldDB; B0L0Y6; -.
DR   SMR; B0L0Y6; -.
DR   GO; GO:0005576; C:extracellular region; IEA:UniProtKB-SubCell.
DR   GO; GO:0090729; F:toxin activity; IEA:UniProtKB-KW.
PE   2: Evidence at transcript level;
KW   Cleavage on pair of basic residues; Disulfide bond; Neurotoxin; Secreted;
KW   Signal; Toxin.
FT   SIGNAL          1..20
FT                   /evidence="ECO:0000255"
FT   PROPEP          21..26
FT                   /id="PRO_0000419829"
FT   CHAIN           29..107
FT                   /note="Putative conotoxin"
FT                   /id="PRO_0000419830"
SQ   SEQUENCE   107 AA;  11509 MW;  9B6C02AE7A08209E CRC64;
     MKLFMFAAII FTMASTTVRA EQCANNRKVC TWDGQGDTNC DCIGTACHED DAHKVSVAGS
     AFYTCQPISA FRVCDGSEDV MDAGFSELYC RCSGGSYTIS NGEVVCD